Keith J. Buckler

Affiliations: 
1993-1999 University Laboratory of Physiology (now DPAG) University of Oxford, Oxford, United Kingdom 
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Pandit JJ, Huskens N, O'Donohoe PB, et al. (2020) Competitive Interactions between Halothane and Isoflurane at the Carotid Body and TASK Channels. Anesthesiology
O'Donohoe PB, Turner PJ, Huskens N, et al. (2018) Influence of propofol on isolated neonatal rat carotid body glomus cell response to hypoxia and hypercapnia. Respiratory Physiology & Neurobiology
O'Donohoe PB, Huskens N, Turner PJ, et al. (2018) A1899, PK-THPP, ML365, and Doxapram inhibit endogenous TASK channels and excite calcium signaling in carotid body type-1 cells. Physiological Reports. 6: e13876
Fielding JW, Hodson EJ, Cheng X, et al. (2018) PHD2 inactivation in Type I cells drives HIF-2α dependent multi-lineage hyperplasia and the formation of paraganglioma-like carotid bodies. The Journal of Physiology
Holmes AP, Turner PJ, Buckler KJ, et al. (2015) Moderate inhibition of mitochondrial function augments carotid body hypoxic sensitivity. Pflugers Archiv : European Journal of Physiology
Hodson EJ, Nicholls LG, Turner PJ, et al. (2015) Regulation of ventilatory sensitivity and carotid body proliferation in hypoxia by the PHD2/HIF-2 pathway. The Journal of Physiology
Buckler KJ, Turner PJ. (2015) Functional Properties of Mitochondria in the Type-1 Cell and Their Role in Oxygen Sensing. Advances in Experimental Medicine and Biology. 860: 69-80
Buckler KJ. (2015) TASK channels in arterial chemoreceptors and their role in oxygen and acid sensing. PflüGers Archiv : European Journal of Physiology. 467: 1013-25
Holmes AP, Turner PJ, Carter P, et al. (2014) Glycogen metabolism protects against metabolic insult to preserve carotid body function during glucose deprivation. The Journal of Physiology. 592: 4493-506
Slingo ME, Turner PJ, Christian HC, et al. (2014) The von Hippel-Lindau Chuvash mutation in mice causes carotid-body hyperplasia and enhanced ventilatory sensitivity to hypoxia. Journal of Applied Physiology (Bethesda, Md. : 1985). 116: 885-92
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