Reid M. Feldman, Ph.D.
Affiliations: | 2000 | California Institute of Technology, Pasadena, CA |
Area:
Ubiquitin-Dependent Proteolysis and Cell DivisionGoogle:
"Reid Feldman"Parents
Sign in to add mentor| Raymond J. Deshaies | grad student | 2000 | Caltech | |
| (Life is degrading. SCF ubiquitin ligases: Their components and functions.) | ||||
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Publications
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| Yoo SJ, Huh JR, Muro I, et al. (2002) Hid, Rpr and Grim negatively regulate DIAP1 levels through distinct mechanisms. Nature Cell Biology. 4: 416-24 |
| Shou W, Sakamoto KM, Keener J, et al. (2001) Net1 stimulates RNA polymerase I transcription and regulates nucleolar structure independently of controlling mitotic exit. Molecular Cell. 8: 45-55 |
| Seol JH, Feldman RM, Zachariae W, et al. (1999) Cdc53/cullin and the essential Hrt1 RING-H2 subunit of SCF define a ubiquitin ligase module that activates the E2 enzyme Cdc34. Genes & Development. 13: 1614-26 |
| Feldman RM, Correll CC, Kaplan KB, et al. (1997) A complex of Cdc4p, Skp1p, and Cdc53p/cullin catalyzes ubiquitination of the phosphorylated CDK inhibitor Sic1p. Cell. 91: 221-30 |
| Verma R, Feldman RM, Deshaies RJ. (1997) SIC1 is ubiquitinated in vitro by a pathway that requires CDC4, CDC34, and cyclin/CDK activities. Molecular Biology of the Cell. 8: 1427-37 |