Daniel T. Starczynowski, Ph.D.
Affiliations: | 2006 | Boston University, Boston, MA, United States |
Area:
Molecular BiologyGoogle:
"Daniel Starczynowski"Parents
Sign in to add mentor| Thomas D. Gilmore | grad student | 2006 | Boston University | |
| (A mutational and functional analysis of C -terminal sequences of human transcription factor REL: Their role in cellular transformation and transcriptional activation.) | ||||
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Publications
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| Venkatasubramanian M, Schwartz L, Ramachandra N, et al. (2024) Broad de-regulated U2AF1 splicing is prognostic and augments leukemic transformation via protein arginine methyltransferase activation. Biorxiv : the Preprint Server For Biology |
| Marion W, Koppe T, Chen CC, et al. (2023) RUNX1 mutations mitigate quiescence to promote transformation of hematopoietic progenitors in Fanconi anemia. Leukemia |
| Dean ST, Ishikawa C, Zhu X, et al. (2023) Repression of TRIM13 by chromatin assembly factor CHAF1B is critical for AML development. Blood Advances |
| Bennett JR, Ishikawa C, Agarwal P, et al. (2023) Paralog-specific signaling by IRAK1/4 maintains MyD88-independent functions in MDS/AML. Blood |
| Barreyro L, Sampson AM, Ishikawa C, et al. (2022) Blocking UBE2N abrogates oncogenic immune signaling in acute myeloid leukemia. Science Translational Medicine. 14: eabb7695 |
| Muto T, Guillamot M, Yeung J, et al. (2022) TRAF6 functions as a tumor suppressor in myeloid malignancies by directly targeting MYC oncogenic activity. Cell Stem Cell |
| Niederkorn M, Ishikawa C, M Hueneman K, et al. (2021) The deubiquitinase USP15 modulates cellular redox and is a therapeutic target in acute myeloid leukemia. Leukemia |
| Schieber M, Marinaccio C, Bolanos LC, et al. (2020) FBXO11 is a candidate tumor suppressor in the leukemic transformation of myelodysplastic syndrome. Blood Cancer Journal. 10: 98 |
| Niederkorn M, Agarwal P, Starczynowski DT. (2020) TIFA and TIFAB: FHA-domain proteins involved in inflammation, hematopoiesis, and disease. Experimental Hematology |
| Muto T, Walker CS, Choi K, et al. (2020) Adaptive response to inflammation contributes to sustained myelopoiesis and confers a competitive advantage in myelodysplastic syndrome HSCs. Nature Immunology |