Christopher M. Counter
Affiliations: | Duke Medical School, Durham, NC, United States |
Website:
http://pharmacology.mc.duke.edu/faculty/counter.htmGoogle:
"Christopher Counter"
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Publications
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| Le Roux Ö, Pershing NLK, Kaltenbrun E, et al. (2022) Genetically manipulating endogenous Kras levels and oncogenic mutations in vivo influences tissue patterning of murine tumorigenesis. Elife. 11 |
| Li S, Counter CM. (2021) Signaling levels mold the RAS mutation tropism of urethane. Elife. 10 |
| Li S, MacAlpine DM, Counter CM. (2020) Capturing the primordial Kras mutation initiating urethane carcinogenesis. Nature Communications. 11: 1800 |
| Adhikari H, Kattan W, Hancock JF, et al. (2020) Abstract 1085: Interrogating the RAS interactome identifies EFR3A as a novel enhancer of RAS oncogenesis Tumor Biology |
| Li S, Balmain A, Counter CM. (2018) A model for RAS mutation patterns in cancers: finding the sweet spot. Nature Reviews. Cancer |
| Fu J, Dang Y, Counter C, et al. (2018) Codon usage regulates human KRAS expression at both transcriptional and translational levels. The Journal of Biological Chemistry |
| Adhikari H, Counter CM. (2018) Interrogating the protein interactomes of RAS isoforms identifies PIP5K1A as a KRAS-specific vulnerability. Nature Communications. 9: 3646 |
| Xu M, Casio M, Range DE, et al. (2018) Copper Chelation as Targeted Therapy in a Mouse Model of Oncogenic BRAF-Driven Papillary Thyroid Cancer. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research |
| Kabiri Z, Greicius G, Zaribafzadeh H, et al. (2018) Wnt signaling suppresses MAPK-driven proliferation of intestinal stem cells. The Journal of Clinical Investigation |
| Sasine JP, Himburg HA, Termini CM, et al. (2018) Wild-type Kras expands and exhausts hematopoietic stem cells. Jci Insight. 3 |