Justin T. Babcock, Ph.D.
Affiliations: | 2012 | Biochemistry and Molecular Biology | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Biochemistry, Cell BiologyGoogle:
"Justin Babcock"Parents
Sign in to add mentor| Lawrence A. Quilliam | grad student | 2012 | Indiana University Bloomington | |
| (mTORC1 contributes to ER stress induced cell death.) | ||||
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Publications
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| Babcock JT, Nguyen HB, He Y, et al. (2013) Mammalian target of rapamycin complex 1 (mTORC1) enhances bortezomib-induced death in tuberous sclerosis complex (TSC)-null cells by a c-MYC-dependent induction of the unfolded protein response. The Journal of Biological Chemistry. 288: 15687-98 |
| Nguyen HB, Babcock JT, Wells CD, et al. (2013) LKB1 tumor suppressor regulates AMP kinase/mTOR-independent cell growth and proliferation via the phosphorylation of Yap. Oncogene. 32: 4100-9 |
| Siroky BJ, Yin H, Babcock JT, et al. (2012) Human TSC-associated renal angiomyolipoma cells are hypersensitive to ER stress. American Journal of Physiology. Renal Physiology. 303: F831-44 |
| Castro AF, Campos T, Babcock JT, et al. (2012) M-Ras induces Ral and JNK activation to regulate MEK/ERK-independent gene expression in MCF-7 breast cancer cells. Journal of Cellular Biochemistry. 113: 1253-64 |
| Nguyen HB, Babcock JT, Wells CD, et al. (2012) Abstract LB-29: LKB1 tumor suppressor regulates AMP kinase/mTOR-independent cell growth and proliferation via the phosphorylation of Yap Cancer Research. 72 |
| Babcock JT, Nguen HB, He Y, et al. (2012) Abstract LB-247: mTORC1 enhances bortezomib-induced death in TSC-null cells by a c-MYC-dependent induction of the unfolded protein response Cancer Research. 72 |
| Babcock JT, Quilliam LA. (2011) Rheb/mTOR activation and regulation in cancer: novel treatment strategies beyond rapamycin. Current Drug Targets. 12: 1223-31 |