Jelena S. Bezbradica, Ph.D.
Affiliations: | 2007 | Vanderbilt University, Nashville, TN |
Area:
ImmunologyGoogle:
"Jelena Bezbradica"Parents
Sign in to add mentorSebastian Joyce | grad student | 2007 | Vanderbilt | |
(Lineage commitment and functional differentiation of invariant natural T (iNKT) cells.) |
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Publications
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Bezbradica JS, Bryant CE. (2023) Inflammasomes as regulators of mechano-immunity. Embo Reports |
Danielli S, Ma Z, Pantazi E, et al. (2023) The ion channel CALHM6 controls bacterial infection-induced cellular cross-talk at the immunological synapse. The Embo Journal. e111450 |
Bezbradica JS, Coll RC, Boucher D. (2022) Activation of the Non-canonical Inflammasome in Mouse and Human Cells. Methods in Molecular Biology (Clifton, N.J.). 2459: 51-63 |
Lee MSJ, Inoue T, Ise W, et al. (2022) B cell-intrinsic TBK1 is essential for germinal center formation during infection and vaccination in mice. The Journal of Experimental Medicine. 219 |
Kumar A, Hill TM, Gordy LE, et al. (2020) Nur77 controls tolerance induction, terminal differentiation, and effector functions in semi-invariant natural killer T cells. Proceedings of the National Academy of Sciences of the United States of America |
McFie M, Koneva L, Collins I, et al. (2020) Ciliary proteins specify the cell inflammatory response by tuning NFκB signaling, independently of primary cilia. Journal of Cell Science |
Bezbradica JS, Joyce S. (2020) NKT Cells Join the Two Step for Inflammasome-Independent IL-1β Release. Cell Reports. 31: 107481 |
Monteleone M, Stanley AC, Chen KW, et al. (2018) Interleukin-1β Maturation Triggers Its Relocation to the Plasma Membrane for Gasdermin-D-Dependent and -Independent Secretion. Cell Reports. 24: 1425-1433 |
Chen KW, Lawlor KE, von Pein JB, et al. (2018) Cutting Edge: Blockade of Inhibitor of Apoptosis Proteins Sensitizes Neutrophils to TNF- but Not Lipopolysaccharide-Mediated Cell Death and IL-1β Secretion. Journal of Immunology (Baltimore, Md. : 1950) |
Boucher D, Monteleone M, Coll RC, et al. (2018) Caspase-1 self-cleavage is an intrinsic mechanism to terminate inflammasome activity. The Journal of Experimental Medicine |