Timothy A. Fields

Affiliations: 
Duke University, Durham, NC 
Area:
Biochemistry
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"Timothy Fields"
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Publications

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Lea WA, Winklhofer T, Zelenchuk L, et al. (2023) Polycystin-1 Interacting Protein-1 (CU062) Interacts with the Ectodomain of Polycystin-1 (PC1). Cells. 12
Sinha S, Dwivedi N, Woodgett J, et al. (2020) Glycogen synthase kinase-3β inhibits tubular regeneration in acute kidney injury by a FoxM1-dependent mechanism. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology
Dwivedi N, Tao S, Jamadar A, et al. (2020) Epithelial Vasopressin Type-2 Receptors Regulate Myofibroblasts by a YAP-CCN2-Dependent Mechanism in Polycystic Kidney Disease. Journal of the American Society of Nephrology : Jasn
Puri RV, Yerrathota S, Home T, et al. (2019) Notch4 activation aggravates NF-kappa B mediated inflammation in HIV-1 associated Nephropathy. Disease Models & Mechanisms
Omede F, Zhang S, Johnson C, et al. (2019) Dietary Phosphate Restriction Attenuates Polycystic Kidney Disease in Mice. American Journal of Physiology. Renal Physiology
Salah SM, Meisenheimer JD, Rao R, et al. (2019) MCP-1 Promotes Detrimental Cardiac Physiology, Pulmonary Edema and Death in the model of Polycystic Kidney Disease. American Journal of Physiology. Renal Physiology
Silva LM, Jacobs DT, Allard BA, et al. (2018) Inhibition of Hedgehog signaling suppresses proliferation and microcyst formation of human Autosomal Dominant Polycystic Kidney Disease cells. Scientific Reports. 8: 4985
Savin VJ, Sharma M, Zhou J, et al. (2015) Renal and Hematological Effects of CLCF-1, a B-Cell-Stimulating Cytokine of the IL-6 Family. Journal of Immunology Research. 2015: 714964
Singh SP, Tao S, Fields TA, et al. (2015) Glycogen synthase kinase-3 inhibition attenuates fibroblast activation and development of fibrosis following renal ischemia-reperfusion in mice. Disease Models & Mechanisms. 8: 931-40
Wang L, Jirka G, Rosenberg PB, et al. (2015) Gq signaling causes glomerular injury by activating TRPC6. The Journal of Clinical Investigation. 125: 1913-26
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