Eileen S. Carpenter, Ph.D.

Affiliations: 
2013 Molecular and Cellular Pharmacology Stony Brook University, Stony Brook, NY, United States 
Area:
Molecular Biology, Oncology
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"Eileen Carpenter"

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Howard C. Crawford grad student 2013 SUNY Stony Brook
 (Defining p110alpha PI3K as a Therapeutic Target in Pancreatic Cancer.)
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Publications

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Yan W, Menjivar RE, Bonilla ME, et al. (2023) Notch signaling regulates immunosuppressive tumor-associated macrophage function in pancreatic cancer. Cancer Immunology Research
Carpenter ES, Kadiyala P, Elhossiny AM, et al. (2023) KRT17High/CXCL8+ tumor cells display both classical and basal features and regulate myeloid infiltration in the pancreatic cancer microenvironment. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research
Menjivar RE, Nwosu ZC, Du W, et al. (2023) Arginase 1 is a key driver of immune suppression in pancreatic cancer. Elife. 12
Yan W, Steele NG, Kemp SB, et al. (2023) Notch signaling regulates immunosuppressive tumor-associated macrophage function in pancreatic cancer. Biorxiv : the Preprint Server For Biology
Du W, Menjivar RE, Donahue KL, et al. (2022) WNT signaling in the tumor microenvironment promotes immunosuppression in murine pancreatic cancer. The Journal of Experimental Medicine. 220
Mello AM, Ngodup T, Lee Y, et al. (2022) Hypoxia promotes an inflammatory phenotype of fibroblasts in pancreatic cancer. Oncogenesis. 11: 56
Kadiyala P, Elhossiny AM, Carpenter ES. (2022) Using Single Cell Transcriptomics to Elucidate the Myeloid Compartment in Pancreatic Cancer. Frontiers in Oncology. 12: 881871
Kremer DM, Nelson BS, Lin L, et al. (2021) GOT1 inhibition promotes pancreatic cancer cell death by ferroptosis. Nature Communications. 12: 4860
Kemp SB, Carpenter ES, Steele NG, et al. (2021) Apolipoprotein E promotes immune suppression in pancreatic cancer through NF-kB-mediated production of CXCL1. Cancer Research
Kemp SB, Steele NG, Carpenter ES, et al. (2021) Pancreatic cancer is marked by complement-high blood monocytes and tumor-associated macrophages. Life Science Alliance. 4
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