Veronique Bourdeau, Ph.D.
Affiliations: | 2000 | Université de Montréal, Montréal, Canada |
Area:
Molecular BiologyGoogle:
"Veronique Bourdeau"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Lea Brakier-Gingras | grad student | 2000 | Université de Montréal | |
| (Identification de nouvelles sequences capables de se replier en differents motifs d'ARN connus.) | ||||
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Publications
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| Parisotto M, Vuong-Robillard N, Kalegari P, et al. (2022) The NAMPT Inhibitor FK866 Increases Metformin Sensitivity in Pancreatic Cancer Cells. Cancers. 14 |
| Del Toro N, Lessard F, Bouchard J, et al. (2021) Cellular Senescence limits Translational Readthrough. Biology Open |
| Igelmann S, Lessard F, Uchenunu O, et al. (2021) A hydride transfer complex reprograms NAD metabolism and bypasses senescence. Molecular Cell. 81: 3848-3865.e19 |
| Saint-Germain E, Mignacca L, Huot G, et al. (2019) Phosphorylation of SOCS1 inhibits the SOCS1-p53 tumor suppressor axis. Cancer Research |
| Del Toro N, Fernandez-Ruiz A, Mignacca L, et al. (2019) Ribosomal protein Rpl22/eL22 regulates the cell cycle by acting as an inhibitor of the CDK4-cyclin D complex. Cell Cycle (Georgetown, Tex.) |
| Deschênes-Simard X, Parisotto M, Rowell MC, et al. (2019) Circumventing senescence is associated with stem cell properties and metformin sensitivity. Aging Cell. e12889 |
| Lessard F, Igelmann S, Trahan C, et al. (2018) Senescence-associated ribosome biogenesis defects contributes to cell cycle arrest through the Rb pathway. Nature Cell Biology |
| McManus FP, Bourdeau V, Acevedo M, et al. (2018) Quantitative SUMO proteomics reveals the modulation of several PML nuclear body associated proteins and an anti-senescence function of UBC9. Scientific Reports. 8: 7754 |
| Ferbeyre G, Parisotto M, Rowell M, et al. (2018) Abstract 1864: Pharmacologic inhibition of NAMPT sensitizes pancreatic cancer cells to the antineoplastic effects of metformin Cancer Research. 78: 1864-1864 |
| Bourdeau V, Ferbeyre G. (2016) CDK4-CDK6 inhibitors induce autophagy-mediated degradation of DNMT1 and facilitate the senescence antitumor response. Autophagy. 0 |