Radhika Gudi, Ph.D.
Affiliations: | 2006 | University of Illinois at Chicago, Health Sciences Center, Chicago, IL 60612, United States |
Area:
Microbiology Biology, Immunology, Oncology, Cell BiologyGoogle:
"Radhika Gudi"Parents
Sign in to add mentor| Prasad Kanteti | grad student | 2006 | University of Illinois at Chicago, Health Sciences Center | |
| (Siva-1 negatively regulates nuclear factor kappa B activity and promotes activation induced cell death.) | ||||
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| Kesarwani P, Chakraborty P, Gudi R, et al. (2016) Blocking TCR restimulation induced necroptosis in adoptively transferred T cells improves tumor control. Oncotarget |
| Gudi R, Haycraft CJ, Bell PD, et al. (2015) Centrobin-mediated regulation of the centrosomal protein 4.1-associated protein (CPAP) level limits centriole length during elongation stage. The Journal of Biological Chemistry. 290: 6890-902 |
| Gudi R, Zou C, Dhar J, et al. (2014) Centrobin-centrosomal protein 4.1-associated protein (CPAP) interaction promotes CPAP localization to the centrioles during centriole duplication. The Journal of Biological Chemistry. 289: 15166-78 |
| Resch U, Schichl YM, Winsauer G, et al. (2009) Siva1 is a XIAP-interacting protein that balances NFkappaB and JNK signalling to promote apoptosis. Journal of Cell Science. 122: 2651-61 |
| Gudi R, Barkinge J, Hawkins S, et al. (2009) Siva-1 promotes K-48 polyubiquitination of TRAF2 and inhibits TCR-mediated activation of NF-kappaB. Journal of Environmental Pathology, Toxicology and Oncology : Official Organ of the International Society For Environmental Toxicology and Cancer. 28: 25-38 |
| Barkinge JL, Gudi R, Sarah H, et al. (2009) The p53-induced Siva-1 plays a significant role in cisplatin-mediated apoptosis. Journal of Carcinogenesis. 8: 2 |
| Gudi R, Barkinge J, Hawkins S, et al. (2006) Siva-1 negatively regulates NF-kappaB activity: effect on T-cell receptor-mediated activation-induced cell death (AICD). Oncogene. 25: 3458-62 |
| Chu F, Barkinge J, Hawkins S, et al. (2005) Expression of Siva-1 protein or its putative amphipathic helical region enhances cisplatin-induced apoptosis in breast cancer cells: effect of elevated levels of BCL-2. Cancer Research. 65: 5301-9 |
| Chu F, Borthakur A, Sun X, et al. (2004) The Siva-1 putative amphipathic helical region (SAH) is sufficient to bind to BCL-XL and sensitize cells to UV radiation induced apoptosis. Apoptosis : An International Journal On Programmed Cell Death. 9: 83-95 |