Stephane Pelletier, Ph.D.
Affiliations: | 2003 | Université de Montréal, Montréal, Canada |
Area:
Pharmacology, Molecular BiologyGoogle:
"Stephane Pelletier"Parents
Sign in to add mentor| Sylvain Meloche | grad student | 2003 | Université de Montréal | |
| (Regulation de la voie Jak /STAT par les recepteurs couples aux proteines G: Role des petites proteines G de la famille Rho.) | ||||
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Publications
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| Rodriguez DA, Quarato G, Liedmann S, et al. (2022) Caspase-8 and FADD prevent spontaneous ZBP1 expression and necroptosis. Proceedings of the National Academy of Sciences of the United States of America. 119: e2207240119 |
| Tummers B, Mari L, Guy CS, et al. (2020) Caspase-8-Dependent Inflammatory Responses Are Controlled by Its Adaptor, FADD, and Necroptosis. Immunity |
| Malireddi RKS, Gurung P, Kesavardhana S, et al. (2020) Innate immune priming in the absence of TAK1 drives RIPK1 kinase activity-independent pyroptosis, apoptosis, necroptosis, and inflammatory disease. The Journal of Experimental Medicine. 217 |
| Shi H, Chapman NM, Wen J, et al. (2019) Amino Acids License Kinase mTORC1 Activity and Treg Cell Function via Small G Proteins Rag and Rheb. Immunity |
| Samir P, Kesavardhana S, Patmore DM, et al. (2019) DDX3X acts as a live-or-die checkpoint in stressed cells by regulating NLRP3 inflammasome. Nature |
| Gingras S, Kuliyev E, Pelletier S. (2017) SCYL1 does not regulate REST expression and turnover. Plos One. 12: e0178680 |
| Karki R, Man SM, Malireddi RK, et al. (2016) NLRC3 is an inhibitory sensor of PI3K-mTOR pathways in cancer. Nature |
| Ippagunta SK, Gangwar R, Finkelstein D, et al. (2016) Keratinocytes contribute intrinsically to psoriasis upon loss of Tnip1 function. Proceedings of the National Academy of Sciences of the United States of America |
| Pelletier S. (2016) SCYL pseudokinases in neuronal function and survival. Neural Regeneration Research. 11: 42-4 |
| Llambi F, Wang YM, Victor B, et al. (2016) BOK Is a Non-canonical BCL-2 Family Effector of Apoptosis Regulated by ER-Associated Degradation. Cell |