Adam J. Case, Ph.D.

Affiliations: 
2011 Free Radical and Radiation Biology University of Iowa, Iowa City, IA 
Area:
Human Development, Immunology, Oncology
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Frederick E. Domann grad student 2011 University of Iowa
 (The role of mitochondrial superoxide in the development of the mammalian hematopoietic system.)
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Publications

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Place TL, Domann FE, Case AJ. (2020) Corrigendum to "Limitations of oxygen delivery to cells in culture: An underappreciated problem in basic and translational research" [Free Radic. Biol. Med. 113 (2017) 311-322]. Free Radical Biology & Medicine
Dickinson K, Case AJ, Kupzyk K, et al. (2020) Exploring Biologic Correlates of Cancer-Related Fatigue in Men With Prostate Cancer: Cell Damage Pathways and Oxidative Stress. Biological Research For Nursing. 1099800420933347
Moshfegh CM, Collins CW, Gunda V, et al. (2019) Mitochondrial superoxide disrupts the metabolic and epigenetic landscape of CD4 and CD8 T-lymphocytes. Redox Biology. 101141
Place TL, Domann FE, Case AJ. (2017) Limitations of Oxygen Delivery to Cells in Culture: An Underappreciated Problem in Basic and Translational Research. Free Radical Biology & Medicine
Case AJ, Tian J, Zimmerman MC. (2016) Increased mitochondrial superoxide in the brain, but not periphery, sensitizes mice to angiotensin II-mediated hypertension. Redox Biology. 11: 82-90
Case AJ, Roessner CT, Tian J, et al. (2016) Mitochondrial Superoxide Signaling Contributes to Norepinephrine-Mediated T-Lymphocyte Cytokine Profiles. Plos One. 11: e0164609
Wang H, Case AJ, Wang WZ, et al. (2016) Redox Signaling and Neural Control of Cardiovascular Function. Oxidative Medicine and Cellular Longevity. 2016: 7086018
Case AJ, Agraz D, Ahmad IM, et al. (2016) Low-Dose Aronia melanocarpa Concentrate Attenuates Paraquat-Induced Neurotoxicity. Oxidative Medicine and Cellular Longevity. 2016: 5296271
Case AJ, Zimmerman MC. (2015) Redox-regulated suppression of splenic T-lymphocyte activation in a model of sympathoexcitation. Hypertension. 65: 916-23
O'Leary BR, Fath MA, Bellizzi AM, et al. (2015) Loss of SOD3 (EcSOD) Expression Promotes an Aggressive Phenotype in Human Pancreatic Ductal Adenocarcinoma. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research. 21: 1741-51
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