Wan-Chi Lin, Ph.D.
Affiliations: | 2011 | University of Nebraska Medical Center, Omaha, NE, United States |
Area:
OncologyGoogle:
"Wan-Chi Lin"Parents
Sign in to add mentor| Kay-Uwe Wagner | grad student | 2011 | University of Nebraska Medical Center | |
| (The Role of c-Myc in Pancreatic Cancer Initiation and Progression.) | ||||
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Publications
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| Nie L, Wei Y, Zhang F, et al. (2019) CDK2-mediated site-specific phosphorylation of EZH2 drives and maintains triple-negative breast cancer. Nature Communications. 10: 5114 |
| Rajbhandari N, Lin WC, Wehde BL, et al. (2017) Autocrine IGF1 Signaling Mediates Pancreatic Tumor Cell Dormancy in the Absence of Oncogenic Drivers. Cell Reports. 18: 2243-2255 |
| Du Y, Yamaguchi H, Wei Y, et al. (2016) Blocking c-Met-mediated PARP1 phosphorylation enhances anti-tumor effects of PARP inhibitors. Nature Medicine |
| Witkiewicz AK, McMillan EA, Balaji U, et al. (2015) Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets. Nature Communications. 6: 6744 |
| Lin WC, Rajbhandari N, Wagner KU. (2014) Cancer cell dormancy in novel mouse models for reversible pancreatic cancer: a lingering challenge in the development of targeted therapies. Cancer Research. 74: 2138-43 |
| Lin WC, Schmidt JW, Creamer BA, et al. (2013) Gain-of-function of Stat5 leads to excessive granulopoiesis and lethal extravasation of granulocytes to the lung. Plos One. 8: e60902 |
| Lin WC, Rajbhandari N, Liu C, et al. (2013) Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. Cancer Research. 73: 1821-30 |
| Lin W, Rajbhandari N, Liu C, et al. (2012) Abstract LB-429: Dormant cancer cells contribute to residual disease in a mouse model for reversible pancreatic cancer Cancer Research. 72 |
| Zhang Q, Sakamoto K, Liu C, et al. (2011) Cyclin D3 compensates for the loss of cyclin D1 during ErbB2-induced mammary tumor initiation and progression. Cancer Research. 71: 7513-24 |
| Lin W, Zhang Q, Opavsky R, et al. (2011) Abstract 2384: A novel mouse model for the study of c-MYC during initiation and progression of pancreatic ductal adenocarcinoma Cancer Research. 71: 2384-2384 |