Melissa J. Parsons, Ph.D.
Affiliations: | 2006 | Emory University, Atlanta, GA |
Area:
Molecular Biology, OncologyGoogle:
"Melissa Parsons"Parents
Sign in to add mentorPaula M. Vertino | grad student | 2006 | Emory | |
(Functional characterization of TMS1 /ASC in apoptotic signaling.) |
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Publications
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Boice AG, Lopez KE, Pandita RK, et al. (2021) Caspase-2 regulates S-phase cell cycle events to protect from DNA damage accumulation independent of apoptosis. Oncogene |
Ando K, Parsons MJ, Shah RB, et al. (2017) NPM1 directs PIDDosome-dependent caspase-2 activation in the nucleolus. The Journal of Cell Biology |
Llambi F, Wang YM, Victor B, et al. (2016) BOK Is a Non-canonical BCL-2 Family Effector of Apoptosis Regulated by ER-Associated Degradation. Cell |
Sanders MG, Parsons MJ, Howard AG, et al. (2015) Single-cell imaging of inflammatory caspase dimerization reveals differential recruitment to inflammasomes. Cell Death & Disease. 6: e1813 |
Ichim G, Lopez J, Ahmed SU, et al. (2015) Limited mitochondrial permeabilization causes DNA damage and genomic instability in the absence of cell death. Molecular Cell. 57: 860-72 |
Parsons MJ, Rehm M, Bouchier-Hayes L. (2015) Imaging-based methods for assessing caspase activity in single cells. Cold Spring Harbor Protocols. 2015: pdb.top070342 |
Garrison SP, Phillips DC, Jeffers JR, et al. (2012) Genetically defining the mechanism of Puma- and Bim-induced apoptosis. Cell Death and Differentiation. 19: 642-9 |
Miller C, Dillon C, Martinez J, et al. (2011) Scientists contemplate unexplained death in Austrian Alps. Embo Molecular Medicine. 3: 363-6 |
Parsons MJ, Green DR. (2010) Mitochondria in cell death. Essays in Biochemistry. 47: 99-114 |
Tait SW, Parsons MJ, Llambi F, et al. (2010) Resistance to caspase-independent cell death requires persistence of intact mitochondria. Developmental Cell. 18: 802-13 |