Naoko Shima

Affiliations: 
Molecular, Cellular, Developmental Biology and Genetics University of Minnesota, Twin Cities, Minneapolis, MN 
Area:
Genetics, Molecular Biology, Cell Biology
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"Naoko Shima"
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Publications

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Leung W, Baxley RM, Traband E, et al. (2023) FANCD2-dependent mitotic DNA synthesis relies on PCNA K164 ubiquitination. Cell Reports. 42: 113523
Traband EL, Hammerlund SR, Shameem M, et al. (2023) Mitotic DNA synthesis in untransformed human cells preserves common fragile site stability via a FANCD2-driven mechanism that requires HELQ. Journal of Molecular Biology. 168294
Graber-Feesl CL, Pederson KD, Aney KJ, et al. (2019) Mitotic DNA synthesis is differentially regulated between cancer and non-cancerous cells. Molecular Cancer Research : McR
Shima N, Pederson KD. (2017) Dormant origins as a built-in safeguard in eukaryotic DNA replication against genome instability and disease development. Dna Repair
Ge XQ, Han J, Cheng EC, et al. (2015) Embryonic Stem Cells License a High Level of Dormant Origins to Protect the Genome against Replication Stress. Stem Cell Reports. 5: 185-94
Luebben SW, Shima N, Kawabata T. (2014) Methods for the detection of genome instability derived from replication stress in primary mouse embryonic fibroblasts. Methods in Molecular Biology (Clifton, N.J.). 1194: 341-52
Luo Y, Hartford SA, Zeng R, et al. (2014) Hypersensitivity of primordial germ cells to compromised replication-associated DNA repair involves ATM-p53-p21 signaling. Plos Genetics. 10: e1004471
Luebben SW, Kawabata T, Johnson CS, et al. (2014) A concomitant loss of dormant origins and FANCC exacerbates genome instability by impairing DNA replication fork progression. Nucleic Acids Research. 42: 5605-15
Luebben SW, Kawabata T, Akre MK, et al. (2013) Helq acts in parallel to Fancc to suppress replication-associated genome instability. Nucleic Acids Research. 41: 10283-97
Kawabata T, Yamaguchi S, Buske T, et al. (2011) A reduction of licensed origins reveals strain-specific replication dynamics in mice. Mammalian Genome : Official Journal of the International Mammalian Genome Society. 22: 506-17
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