Marsha D. Wallace, Ph.D.

Affiliations: 
2012 Cornell University, Ithaca, NY, United States 
Area:
Genetics, Oncology
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"Marsha Wallace"

Parents

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John Schimenti grad student 2012 Cornell
 (Elucidation of mechanisms in mammary tumorigenesis and identification of driving events and susceptibility loci.)
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Publications

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Zhang P, Kitchen-Smith I, Xiong L, et al. (2021) Germline and somatic genetic variants in the p53 pathway interact to affect cancer risk, progression, and drug response. Cancer Research
Di Giovannantonio M, Harris BH, Zhang P, et al. (2020) Heritable genetic variants in key cancer genes link cancer risk with anthropometric traits. Journal of Medical Genetics
Surakhy M, Wallace M, Bond E, et al. (2020) A common polymorphism in the retinoic acid pathway modifies adrenocortical carcinoma age-dependent incidence. British Journal of Cancer
McNairn AJ, Chuang CH, Bloom JC, et al. (2019) Female-biased embryonic death from inflammation induced by genomic instability. Nature
Kartha N, Shen L, Maskin C, et al. (2016) The Chromatin Remodeling Component Arid1a Is a Suppressor of Spontaneous Mammary Tumors in Mice. Genetics
Stracquadanio G, Wang X, Wallace MD, et al. (2016) The importance of p53 pathway genetics in inherited and somatic cancer genomes. Nature Reviews. Cancer. 16: 251-65
Stracquadanio G, Wallace M, Grawenda A, et al. (2016) Polymorphisms in the p53 pathway are enriched in cancer susceptibility loci and share characteristics with somatic pathway mutations European Journal of Cancer. 61: S179
Wallace MD, Southard TL, Schimenti KJ, et al. (2014) Role of DNA damage response pathways in preventing carcinogenesis caused by intrinsic replication stress. Oncogene. 33: 3688-95
Wallace MD, Pfefferle AD, Shen L, et al. (2012) Comparative oncogenomics implicates the neurofibromin 1 gene (NF1) as a breast cancer driver. Genetics. 192: 385-96
Kawabata T, Yamaguchi S, Buske T, et al. (2011) A reduction of licensed origins reveals strain-specific replication dynamics in mice. Mammalian Genome : Official Journal of the International Mammalian Genome Society. 22: 506-17
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