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James J. Tomasek

Affiliations: 
1975-1980 Biological Sciences State University of New York, Albany, Albany, NY, United States 
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"James Tomasek"
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Parents

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Stuart A. Newman grad student 1975-1980 SUNY Albany
Elizabeth D. Hay post-doc Harvard Medical School

Children

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Melville B Vaughan research assistant 1989-1993 University of Oklahoma Health Sciences Center
Brian K. Pilcher grad student 1989-1994 University of Oklahoma Health Sciences Center
Melville B Vaughan grad student 1993-1998 University of Oklahoma Health Sciences Center

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Publications

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Ge Y, Weygant N, Qu D, et al. (2018) Alternative splice variants of DCLK1 mark cancer stem cells, promote self-renewal and drug-resistance, and can be targeted to inhibit tumorigenesis in kidney cancer. International Journal of Cancer
Bergmeier V, Etich J, Pitzler L, et al. (2017) Identification of a myofibroblast-specific expression signature in skin wounds. Matrix Biology : Journal of the International Society For Matrix Biology
Qu D, Weygant N, Berry WL, et al. (2017) Abstract 3888: DCLK1 is part of an EMT feedback loop and promotes colorectal cancer cell invasion and drug resistance Cancer Research. 77: 3888-3888
Ge Y, Weygant N, Qu D, et al. (2016) Abstract 3340: Doublecortin-like kinase 1 marks cancer stem-like cells and modulates drug-resistance, self-renewal, and tumorigenesis in renal cancer Cancer Research. 76: 3340-3340
Qu D, Weygant N, May R, et al. (2016) Abstract 1731: Overexpression of DCLK1 in pancreatic cancer activates KRAS/PI3K/MTOR pathway signaling and supports tumorigenesis, invasiveness, and stemness Cancer Research. 76: 1731-1731
Weygant N, Qu D, May R, et al. (2015) DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinoma. Oncotarget. 6: 2193-205
Weygant N, Qu D, May R, et al. (2015) Abstract 4374: DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinoma Cancer Research. 75: 4374-4374
McBride JD, Jenkins AJ, Liu X, et al. (2014) Elevated circulation levels of an antiangiogenic SERPIN in patients with diabetic microvascular complications impair wound healing through suppression of Wnt signaling. The Journal of Investigative Dermatology. 134: 1725-34
Van De Water L, Varney S, Tomasek JJ. (2013) Mechanoregulation of the Myofibroblast in Wound Contraction, Scarring, and Fibrosis: Opportunities for New Therapeutic Intervention. Advances in Wound Care. 2: 122-141
Tomasek JJ, Haaksma CJ, Schwartz RJ, et al. (2013) Whole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transition. Wound Repair and Regeneration : Official Publication of the Wound Healing Society [and] the European Tissue Repair Society. 21: 166-76
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