Erin A. Nekritz
Affiliations: | University of California, San Francisco, San Francisco, CA |
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"Erin Nekritz"
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Kumar P, Koach J, Nekritz E, et al. (2024) Aurora Kinase A inhibition enhances DNA damage and tumor cell death with 131I-MIBG therapy in high-risk neuroblastoma. Research Square |
Rodriguez-Barrueco R, Nekritz EA, Bertucci F, et al. (2017) miR-424(322)/503 is a breast cancer tumor suppressor whose loss promotes resistance to chemotherapy. Genes & Development. 31: 553-566 |
Temple W, Mendelsohn L, Kim GE, et al. (2016) Erratum to: Vesicular monoamine transporter protein expression correlates with clinical features, tumor biology, and MIBG avidity in neuroblastoma: a report from the Children's Oncology Group. European Journal of Nuclear Medicine and Molecular Imaging |
Temple W, Mendelsohn L, Kim GE, et al. (2015) Vesicular monoamine transporter protein expression correlates with clinical features, tumor biology, and MIBG avidity in neuroblastoma: a report from the Children's Oncology Group. European Journal of Nuclear Medicine and Molecular Imaging |
DuBois SG, Temple W, Mendelsohn L, et al. (2015) Vesicular monoamine transporter protein expression in neuroblastoma: A report from the Children’s Oncology Group. Journal of Clinical Oncology. 33: 10043-10043 |
Gustafson WC, Meyerowitz JG, Nekritz EA, et al. (2014) Drugging MYCN through an allosteric transition in Aurora kinase A. Cancer Cell. 26: 414-27 |
Gustafson WC, Meyerowitz JG, Nekritz EA, et al. (2014) Abstract PR08: Drugging MYCN protein stability through an allosteric transition in Aurora kinase A Cancer Research. 74 |
Meyerowitz JG, Gustafson WC, Nekritz E, et al. (2014) Abstract B78: Targeting the translational apparatus in MYCN-driven medulloblastoma Cancer Research. 74 |
Gustafson WC, Meyerowitz JG, Nekritz EA, et al. (2014) Abstract 2688: Drugging MYCN through an allosteric transition in Aurora A kinase Cancer Research. 74: 2688-2688 |
Puissant A, Frumm SM, Alexe G, et al. (2013) Targeting MYCN in neuroblastoma by BET bromodomain inhibition Cancer Discovery. 3: 309-323 |