BETA: Related publications
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| Williams C, Brown R, Zhao Y, et al. (2023) MTGR1 is required to maintain small intestinal stem cell populations. Research Square |
| Brown RE, Anant SA, Parang B, et al. (2020) 1108 A SINGLE RESIDUE IN MYELOID TRANSLOCATION GENE ON CHROMOSOME 16 (MTG16P209T) REGULATES EPITHELIAL RESPONSE TO INJURY AND INFLAMMATORY TUMORIGENESIS Gastroenterology. 158: S-215-S-216 |
| Thompson JJ, Short SP, Parang B, et al. (2019) Blood vessel epicardial substance (BVES) reduces LRP6 receptor and cytoplasmic -catenin levels to modulate Wnt signaling and intestinal homeostasis. Carcinogenesis |
| Brown RE, Parang B, Short SP, et al. (2019) 993 – Myeloid Translocation Gene on Chromosome 16 (MTG16) is a Transcriptional Co-Repressor that Regulates Epithelial Response to Injury Via Homology Regions Nhr1 and Nhr3 Gastroenterology. 156: S-211 |
| Parang B, Thompson JJ, Williams CS. (2018) Blood Vessel Epicardial Substance (BVES) in junctional signaling and cancer. Tissue Barriers. 1-12 |
| Choksi YA, Reddy VK, Singh K, et al. (2018) BVES is required for maintenance of colonic epithelial integrity in experimental colitis by modifying intestinal permeability. Mucosal Immunology |
| Short SP, Parang B, Choksi YA, et al. (2018) 595 - MTGR1 Restricts the Intestinal Stem Cell Compartment and Protects from Stress Induced Failure Gastroenterology. 154: S-126 |
| McDonough EM, Barrett CW, Parang B, et al. (2017) MTG16 is a tumor suppressor in colitis-associated carcinoma. Jci Insight. 2 |
| Parang B, Kaz AM, Barrett CW, et al. (2017) BVES regulates c-Myc stability via PP2A and suppresses colitis-induced tumourigenesis. Gut. 66: 852-862 |
| Parang B, Bradley AM, Mittal MK, et al. (2016) Myeloid translocation genes differentially regulate colorectal cancer programs. Oncogene |