BETA: Related publications
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| Pilat JM, Brown RE, Chen Z, et al. (2023) SELENOP modifies sporadic colorectal carcinogenesis and WNT signaling activity through LRP5/6 interactions. The Journal of Clinical Investigation |
| Pilat JM, Li C, Thompson JJ, et al. (2020) Sa1186 SELENOPROTEIN P (SELENOP) AS A CONTEXT-DEPENDENT WNT SIGNALING MODULATOR IN COLORECTAL CANCER CELLS Gastroenterology. 158 |
| Fei-Zhang DJ, Thompson JJ, Frey MR, et al. (2020) Sa1181 BCAR3 PARTNERS WITH THE EGF-RECEPTOR TYROSINE KINASE THROUGH ITS SH2 DOMAIN TO PROMOTE COLORECTAL CANCER CELL INVASIVENESS Gastroenterology. 158 |
| Wimbiscus M, Aziz Z, Chaparro J, et al. (2020) Gpx3 is Reduced in Eosinophilic Esophagitis Patients and Regulates Esophageal Epithelial Homeostasis Free Radical Biology and Medicine. 159: S40 |
| Thompson JJ, Short SP, Parang B, et al. (2019) Blood vessel epicardial substance (BVES) reduces LRP6 receptor and cytoplasmic -catenin levels to modulate Wnt signaling and intestinal homeostasis. Carcinogenesis |
| Short SP, Thompson JJ, Bilotta AJ, et al. (2019) Serine Threonine Kinase 17A maintains the epithelial state in colorectal cancer cells. Molecular Cancer Research : McR |
| Pilat JM, Short SP, Thompson JJ, et al. (2019) Sa1708 – Selenoprotein P Modifies Wnt-Driven Sporadic Colorectal Carcinogenesis Gastroenterology. 156 |
| Brown RE, Parang B, Short SP, et al. (2019) 993 – Myeloid Translocation Gene on Chromosome 16 (MTG16) is a Transcriptional Co-Repressor that Regulates Epithelial Response to Injury Via Homology Regions Nhr1 and Nhr3 Gastroenterology. 156: S-211 |
| Parang B, Thompson JJ, Williams CS. (2018) Blood Vessel Epicardial Substance (BVES) in junctional signaling and cancer. Tissue Barriers. 1-12 |
| Choksi YA, Reddy VK, Singh K, et al. (2018) BVES is required for maintenance of colonic epithelial integrity in experimental colitis by modifying intestinal permeability. Mucosal Immunology |