Fan-E Mo, Ph.D. - Publications

Affiliations: 
2000 University of Illinois at Chicago, Health Sciences Center, Chicago, IL 60612, United States 
Area:
Genetics, Obstetrics and Gynecology

13 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Wang YK, Weng HK, Mo FE. The regulation and functions of the matricellular CCN proteins induced by shear stress. Journal of Cell Communication and Signaling. PMID 37191841 DOI: 10.1007/s12079-023-00760-z  0.339
2021 Mo FE. Shear-Regulated Extracellular Microenvironments and Endothelial Cell Surface Integrin Receptors Intertwine in Atherosclerosis. Frontiers in Cell and Developmental Biology. 9: 640781. PMID 33889574 DOI: 10.3389/fcell.2021.640781  0.435
2019 Mo FE, Hsu PL. Response by Mo and Hsu to Letter Regarding Article, "Shear-Induced CCN1 Promotes Atheroprone Endothelial Phenotypes and Atherosclerosis". Circulation. 140: e768-e769. PMID 31710519 DOI: 10.1161/CIRCULATIONAHA.119.043154  0.32
2019 Su BC, Hsu PL, Mo FE. CCN1 triggers adaptive autophagy in cardiomyocytes to curb its apoptotic activities. Journal of Cell Communication and Signaling. PMID 31659628 DOI: 10.1007/s12079-019-00534-6  0.317
2019 Hsu PL, Chen JS, Wang CY, Wu HL, Mo FE. Shear-Induced CCN1 Promotes Atheroprone Endothelial Phenotypes and Atherosclerosis. Circulation. PMID 30917686 DOI: 10.1161/Circulationaha.118.033895  0.422
2017 Mo FE, Hsu PL. The extracellular matrix protein CCN1 mediates the endothelial dysfunction induced by disturbed flow. Atherosclerosis. 263: e32-e33. PMID 29366078 DOI: 10.1016/j.atherosclerosis.2017.06.125  0.373
2016 Hsu PL, Mo FE. Matricellular protein CCN1 mediates doxorubicin-induced cardiomyopathy in mice. Oncotarget. PMID 27167338 DOI: 10.18632/oncotarget.9162  0.303
2013 Hsu PL, Su BC, Kuok QY, Mo FE. Extracellular matrix protein CCN1 regulates cardiomyocyte apoptosis in mice with stress-induced cardiac injury. Cardiovascular Research. 98: 64-72. PMID 23329650 DOI: 10.1093/cvr/cvt001  0.387
2006 Mo FE, Lau LF. The matricellular protein CCN1 is essential for cardiac development. Circulation Research. 99: 961-9. PMID 17023674 DOI: 10.1161/01.Res.0000248426.35019.89  0.583
2002 Mo FE, Muntean AG, Chen CC, Stolz DB, Watkins SC, Lau LF. CYR61 (CCN1) is essential for placental development and vascular integrity. Molecular and Cellular Biology. 22: 8709-20. PMID 12446788 DOI: 10.1128/Mcb.22.24.8709-8720.2002  0.618
2001 Chen CC, Mo FE, Lau LF. The angiogenic factor Cyr61 activates a genetic program for wound healing in human skin fibroblasts. The Journal of Biological Chemistry. 276: 47329-37. PMID 11584015 DOI: 10.1074/Jbc.M107666200  0.606
2001 Latinkic BV, Mo FE, Greenspan JA, Copeland NG, Gilbert DJ, Jenkins NA, Ross SR, Lau LF. Promoter function of the angiogenic inducer Cyr61gene in transgenic mice: tissue specificity, inducibility during wound healing, and role of the serum response element. Endocrinology. 142: 2549-57. PMID 11356704 DOI: 10.1210/Endo.142.6.8208  0.671
1996 Kireeva ML, MO FE, Yang GP, Lau LF. Cyr61, a product of a growth factor-inducible immediate-early gene, promotes cell proliferation, migration, and adhesion. Molecular and Cellular Biology. 16: 1326-34. PMID 8657105 DOI: 10.1128/Mcb.16.4.1326  0.658
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