| Year |
Citation |
Score |
| 2023 |
Li K, Kratzmann V, Dai M, Gatzke N, Rocic P, Bramlage P, Grisk O, Lubomirov LT, Hoffmeister M, Lauxmann MA, Ritter O, Buschmann E, Bader M, Persson AB, Buschmann I, et al. Angiotensin receptor-neprilysin inhibitor improves coronary collateral perfusion. Frontiers in Cardiovascular Medicine. 9: 981333. PMID 36818914 DOI: 10.3389/fcvm.2022.981333 |
0.326 |
|
| 2021 |
Dhagia V, Kitagawa A, Jacob C, Zheng C, D'Alessandro A, Edwards JG, Rocic P, Gupte R, Gupte SA. G6PD activity contributes to the regulation of histone acetylation and gene expression in smooth muscle cells and to the pathogenesis of vascular diseases. American Journal of Physiology. Heart and Circulatory Physiology. PMID 33416454 DOI: 10.1152/ajpheart.00488.2020 |
0.341 |
|
| 2020 |
Kitagawa A, Kizub I, Jacob C, Michael K, D'Alessandro A, Reisz JA, Grzybowski M, Geurts AM, Rocic P, Gupte R, Miano JM, Gupte SA. CRISPR-Mediated Single Nucleotide Polymorphism Modeling in Rats Reveals Insight Into Reduced Cardiovascular Risk Associated With Mediterranean Variant. Hypertension (Dallas, Tex. : 1979). HYPERTENSIONAHA12014. PMID 32507041 DOI: 10.1161/Hypertensionaha.120.14772 |
0.417 |
|
| 2020 |
Gupte R, Dhagia V, Rocic P, Ochi R, Gupte SA. Glucose-6-phosphate dehydrogenase increases Ca currents by interacting with Ca1.2 and reducing intrinsic inactivation of the L-type calcium channel. American Journal of Physiology. Heart and Circulatory Physiology. PMID 32442021 DOI: 10.1152/Ajpheart.00727.2019 |
0.339 |
|
| 2020 |
Signorelli SS, Vanella L, Abraham NG, Scuto S, Marino E, Rocic P. Pathophysiology of chronic peripheral ischemia: new perspectives. Therapeutic Advances in Chronic Disease. 11: 2040622319894466. PMID 32076496 DOI: 10.1177/2040622319894466 |
0.339 |
|
| 2019 |
Rocic P. Comparison of Cardiovascular Benefits of Bariatric Surgery and Abdominal Lipectomy. Current Hypertension Reports. 21: 37. PMID 30953254 DOI: 10.1007/S11906-019-0945-8 |
0.303 |
|
| 2018 |
Rocic P, Schwartzman ML. 20-HETE in the regulation of vascular and cardiac function. Pharmacology & Therapeutics. PMID 30048707 DOI: 10.1016/J.Pharmthera.2018.07.004 |
0.379 |
|
| 2018 |
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, ... Rocic P, et al. Corrigendum to "Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation" [J. Mol. Cell. Cardiol. 117 (2018) 88-99]. Journal of Molecular and Cellular Cardiology. PMID 29625729 DOI: 10.1016/J.Yjmcc.2018.03.015 |
0.77 |
|
| 2018 |
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, ... Rocic P, et al. Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation. Journal of Molecular and Cellular Cardiology. PMID 29428638 DOI: 10.1016/J.Yjmcc.2018.02.005 |
0.773 |
|
| 2017 |
Hunter I, Soler A, Joseph G, Hutcheson B, Bradford C, Zhang F, Potter BJ, Proctor SD, Rocic P. Cardiovascular function in male and female JCR:LA-cp rats: Effect of high fat/high sucrose diet. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00535.2016. PMID 28087518 DOI: 10.1152/Ajpheart.00535.2016 |
0.416 |
|
| 2016 |
Joseph G, Soler A, Hutcheson R, Hunter I, Bradford C, Hutcheson B, Gotlinger KH, Jiang H, Falck JR, Proctor S, Laniado Schwartzman M, Rocic P. Elevated 20-HETE Impairs Coronary Collateral Growth in Metabolic Syndrome Via Endothelial Dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00561.2016. PMID 28011587 DOI: 10.1152/Ajpheart.00561.2016 |
0.767 |
|
| 2016 |
Diane A, Pierce WD, Kelly SE, Sokolik S, Borthwick F, Jacome-Sosa M, Mangat R, Pradillo JM, Allan SM, Ruth MR, Field CJ, Hutcheson R, Rocic P, Russell JC, Vine DF, et al. Mechanisms of Comorbidities Associated With the Metabolic Syndrome: Insights from the JCR:LA-cp Corpulent Rat Strain. Frontiers in Nutrition. 3: 44. PMID 27777929 DOI: 10.3389/Fnut.2016.00044 |
0.737 |
|
| 2016 |
Rocic P. Can microRNAs Be Biomarkers or Targets for Therapy of Ischemic Coronary Artery Disease in Metabolic Syndrome? Current Drug Targets. PMID 26844562 DOI: 10.2174/1389450117666160201113734 |
0.461 |
|
| 2015 |
Hutcheson R, Terry R, Hutcheson B, Jadhav R, Chaplin J, Smith E, Barrington R, Proctor SD, Rocic P. miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 308: H1323-35. PMID 25840830 DOI: 10.1152/Ajpheart.00654.2014 |
0.749 |
|
| 2015 |
Rocic P, Gupte S. The Role of Vascular Smooth Muscle Phenotype in Coronary Artery Disease Translational Research in Coronary Artery Disease: Pathophysiology to Treatment. 15-22. DOI: 10.1016/B978-0-12-802385-3.00002-4 |
0.329 |
|
| 2014 |
Villalta PC, Rocic P, Townsley MI. Role of MMP2 and MMP9 in TRPV4-induced lung injury. American Journal of Physiology. Lung Cellular and Molecular Physiology. 307: L652-9. PMID 25150065 DOI: 10.1152/Ajplung.00113.2014 |
0.327 |
|
| 2014 |
Hutcheson R, Chaplin J, Hutcheson B, Borthwick F, Proctor S, Gebb S, Jadhav R, Smith E, Russell JC, Rocic P. miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 4088-99. PMID 24903275 DOI: 10.1096/Fj.14-251223 |
0.793 |
|
| 2014 |
Leavesley SJ, Ledkins W, Rocic P. A device for performing automated balloon catheter inflation ischemia studies. Plos One. 9: e95823. PMID 24769885 DOI: 10.1371/Journal.Pone.0095823 |
0.431 |
|
| 2013 |
Dodd T, Wiggins L, Hutcheson R, Smith E, Musiyenko A, Hysell B, Russell JC, Rocic P. Impaired coronary collateral growth in the metabolic syndrome is in part mediated by matrix metalloproteinase 12-dependent production of endostatin and angiostatin. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 1339-49. PMID 23599440 DOI: 10.1161/Atvbaha.113.301533 |
0.74 |
|
| 2013 |
Alzoubi A, Toba M, Abe K, O'Neill KD, Rocic P, Fagan KA, McMurtry IF, Oka M. Dehydroepiandrosterone restores right ventricular structure and function in rats with severe pulmonary arterial hypertension. American Journal of Physiology. Heart and Circulatory Physiology. 304: H1708-18. PMID 23585128 DOI: 10.1152/Ajpheart.00746.2012 |
0.317 |
|
| 2013 |
Hutcheson R, Terry R, Chaplin J, Smith E, Musiyenko A, Russell JC, Lincoln T, Rocic P. MicroRNA-145 restores contractile vascular smooth muscle phenotype and coronary collateral growth in the metabolic syndrome. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 727-36. PMID 23393394 DOI: 10.1161/Atvbaha.112.301116 |
0.754 |
|
| 2012 |
Hutcheson R, Rocic P. The metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration. Experimental Diabetes Research. 2012: 271028. PMID 22829804 DOI: 10.1155/2012/271028 |
0.75 |
|
| 2012 |
Rocic P. Why is coronary collateral growth impaired in type II diabetes and the metabolic syndrome? Vascular Pharmacology. 57: 179-86. PMID 22342811 DOI: 10.1016/J.Vph.2012.02.001 |
0.504 |
|
| 2012 |
Pung YF, Rocic P, Murphy MP, Smith RA, Hafemeister J, Ohanyan V, Guarini G, Yin L, Chilian WM. Resolution of mitochondrial oxidative stress rescues coronary collateral growth in Zucker obese fatty rats. Arteriosclerosis, Thrombosis, and Vascular Biology. 32: 325-34. PMID 22155454 DOI: 10.1161/Atvbaha.111.241802 |
0.455 |
|
| 2011 |
Dodd T, Jadhav R, Wiggins L, Stewart J, Smith E, Russell JC, Rocic P. MMPs 2 and 9 are essential for coronary collateral growth and are prominently regulated by p38 MAPK. Journal of Molecular and Cellular Cardiology. 51: 1015-25. PMID 21884701 DOI: 10.1016/J.Yjmcc.2011.08.012 |
0.755 |
|
| 2011 |
Torres RA, Drake DA, Solodushko V, Jadhav R, Smith E, Rocic P, Weber DS. Slingshot isoform-specific regulation of cofilin-mediated vascular smooth muscle cell migration and neointima formation. Arteriosclerosis, Thrombosis, and Vascular Biology. 31: 2424-31. PMID 21868701 DOI: 10.1161/Atvbaha.111.232769 |
0.356 |
|
| 2011 |
Perez J, Torres RA, Rocic P, Cismowski MJ, Weber DS, Darley-Usmar VM, Lucchesi PA. PYK2 signaling is required for PDGF-dependent vascular smooth muscle cell proliferation. American Journal of Physiology. Cell Physiology. 301: C242-51. PMID 21451101 DOI: 10.1152/Ajpcell.00315.2010 |
0.308 |
|
| 2011 |
Jadhav R, Dodd T, Smith E, Bailey E, Delucia AL, Russell JC, Madison R, Potter B, Walsh K, Jo H, Rocic P. Angiotensin type I receptor blockade in conjunction with enhanced Akt activation restores coronary collateral growth in the metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 300: H1938-49. PMID 21335466 DOI: 10.1152/Ajpheart.00282.2010 |
0.781 |
|
| 2009 |
Rocic P. Differential phosphoinositide 3-kinase signaling: implications for PTCA? American Journal of Physiology. Heart and Circulatory Physiology. 297: H1970-1. PMID 19837947 DOI: 10.1152/Ajpheart.00952.2009 |
0.384 |
|
| 2009 |
Carrão AC, Chilian WM, Yun J, Kolz C, Rocic P, Lehmann K, van den Wijngaard JP, van Horssen P, Spaan JA, Ohanyan V, Pung YF, Buschmann I. Stimulation of coronary collateral growth by granulocyte stimulating factor: role of reactive oxygen species. Arteriosclerosis, Thrombosis, and Vascular Biology. 29: 1817-22. PMID 19542022 DOI: 10.1161/Atvbaha.109.186445 |
0.354 |
|
| 2009 |
Yun J, Rocic P, Pung YF, Belmadani S, Carrao AC, Ohanyan V, Chilian WM. Redox-dependent mechanisms in coronary collateral growth: the "redox window" hypothesis. Antioxidants & Redox Signaling. 11: 1961-74. PMID 19416057 DOI: 10.1089/Ars.2009.2476 |
0.374 |
|
| 2009 |
Reed R, Potter B, Smith E, Jadhav R, Villalta P, Jo H, Rocic P. Redox-sensitive Akt and Src regulate coronary collateral growth in metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 296: H1811-21. PMID 19376806 DOI: 10.1152/Ajpheart.00920.2008 |
0.494 |
|
| 2008 |
Reed R, Kolz C, Potter B, Rocic P. The mechanistic basis for the disparate effects of angiotensin II on coronary collateral growth. Arteriosclerosis, Thrombosis, and Vascular Biology. 28: 61-7. PMID 17962624 DOI: 10.1161/Atvbaha.107.154294 |
0.318 |
|
| 2007 |
Saitoh S, Kiyooka T, Rocic P, Rogers PA, Zhang C, Swafford A, Dick GM, Viswanathan C, Park Y, Chilian WM. Redox-dependent coronary metabolic dilation. American Journal of Physiology. Heart and Circulatory Physiology. 293: H3720-5. PMID 17965288 DOI: 10.1152/Ajpheart.00436.2007 |
0.333 |
|
| 2007 |
Hattan N, Chilian WM, Park F, Rocic P. Restoration of coronary collateral growth in the Zucker obese rat: impact of VEGF and ecSOD. Basic Research in Cardiology. 102: 217-23. PMID 17323199 DOI: 10.1007/S00395-007-0646-3 |
0.477 |
|
| 2007 |
Rocic P, Kolz C, Reed R, Potter B, Chilian WM. Optimal reactive oxygen species concentration and p38 MAP kinase are required for coronary collateral growth. American Journal of Physiology. Heart and Circulatory Physiology. 292: H2729-36. PMID 17308014 DOI: 10.1152/Ajpheart.01330.2006 |
0.411 |
|
| 2007 |
Chilian WM, Rocic P. Mechanisms Underlying Coronary Collateral Growth The Faseb Journal. 21. DOI: 10.1096/Fasebj.21.5.A79-D |
0.393 |
|
| 2007 |
Rocic P, Reed RE, Kolz C. Redox-Dependent Effects of Angiotensin II on Coronary Collateral Growth The Faseb Journal. 21. DOI: 10.1096/Fasebj.21.5.A527-D |
0.399 |
|
| 2005 |
Rocic P, Lucchesi PA. NAD(P)H oxidases and TGF-beta-induced cardiac fibroblast differentiation: Nox-4 gets Smad. Circulation Research. 97: 850-2. PMID 16254216 DOI: 10.1161/01.Res.0000190403.87462.Bf |
0.338 |
|
| 2005 |
Toyota E, Warltier DC, Brock T, Ritman E, Kolz C, O'Malley P, Rocic P, Focardi M, Chilian WM. Vascular endothelial growth factor is required for coronary collateral growth in the rat. Circulation. 112: 2108-13. PMID 16203926 DOI: 10.1161/Circulationaha.104.526954 |
0.396 |
|
| 2005 |
Koshida R, Rocic P, Saito S, Kiyooka T, Zhang C, Chilian WM. Role of focal adhesion kinase in flow-induced dilation of coronary arterioles. Arteriosclerosis, Thrombosis, and Vascular Biology. 25: 2548-53. PMID 16195476 DOI: 10.1161/01.Atv.0000188511.84138.9B |
0.329 |
|
| 2002 |
Dell'Italia LJ, Rocic P, Lucchesi PA. Use of angiotensin-converting enzyme inhibitors in patients with diabetes and coronary artery disease. Current Problems in Cardiology. 27: 6-36. PMID 11815752 DOI: 10.1067/Mcd.2002.121580 |
0.351 |
|
| 2002 |
Rocic P, Griffin TM, McRae CN, Lucchesi PA. Altered PYK2 phosphorylation by ANG II in hypertensive vascular smooth muscle. American Journal of Physiology. Heart and Circulatory Physiology. 282: H457-65. PMID 11788392 DOI: 10.1152/Ajpheart.00546.2001 |
0.369 |
|
| 2001 |
Rocic P, Lucchesi PA. Down-regulation by Antisense Oligonucleotides Establishes a Role for the Proline-rich Tyrosine Kinase PYK2 in Angiotensin II-induced Signaling in Vascular Smooth Muscle Journal of Biological Chemistry. 276: 21902-21906. PMID 11262415 DOI: 10.1074/Jbc.M101684200 |
0.322 |
|
| 2001 |
Rocic P, Govindarajan G, Sabri A, Lucchesi PA. A role for PYK2 in regulation of ERK1/2 MAP kinases and PI 3-kinase by ANG II in vascular smooth muscle American Journal of Physiology - Cell Physiology. 280. PMID 11121380 DOI: 10.1152/Ajpcell.2001.280.1.C90 |
0.303 |
|
| Show low-probability matches. |